Mazda Jenab, PhD, is a Canadian Scientist serving as Deputy Branch Head in the Nutrition and Metabolism Branch and co-lead of the Onco-Metabolomics Team at the International Agency for Research on Cancer (IARC) in Lyon, France. He obtained his PhD in Nutritional Sciences from the University of Toronto, Canada.

Dr. Jenab’s research focuses on molecular and metabolic epidemiology, with an emphasis on understanding how diet, metabolism, and lifestyle factors contribute to the development of cancer. His work spans multiple domains, including metabolomics, metabolic dysfunction, dietary exposures, microbiomerelated carcinogenesis, and metabolic pathways linking obesity and chronic disease. His scientific contributions are deeply embedded in large international cohorts such as the European Prospective Investigation into Cancer and Nutrition (EPIC), where he has played a central role in evaluating metabolic markers, dietary risk factors, and cancer outcomes, with a focus on colorectal, hepatobiliary and other cancers of the digestive tract.

A highly cited researcher, Dr. Jenab has authored or coauthored over 300 scientific publications, with more than 46,000 citations and an hindex exceeding 100, reflecting his influential contributions to the fields of cancer epidemiology and metabolic research. His work has appeared in leading journals including the BMJ, International Journal of Cancer, BMC Medicine, and Hepatology. Recent publications explore topics such as metabolic biomarkers and mortality in colorectal cancer, gastric cancer metabolomics, microbiome dysbiosis and hepatobiliary cancers, and dietary contaminant exposures.

In addition to his research, Dr. Jenab contributes to international scientific training, serving as a co-Director of the IARC Epidemiology Summer School and as a lecturer in IARC’s educational modules on cancer prevention, obesity, and global public health. His collaborative work with global partners—including scientists from across Europe, North America, and Asia—continues to advance understanding of how molecular nutrition and metabolic perturbations shape cancer risk across populations.